Publications

(2025). The m6A-binding protein YTHDF3 modulates the cardiac response to stress. RNA, 31(7), pp. 923–932.
(2025). The cardiac METTL3/m6A pathway regulates the systemic response to Western diet. JCI Insight, 10(11), pp. e188414.
(2025). AIMP3 maintains cardiac homeostasis by regulating the editing activity of methionyl-tRNA synthetase. Nature Cardiovascular Research, 4(7), pp. 876–890.
(2024). YTHDF2 governs muscle size through a targeted modulation of proteostasis. Nature Communications, 15(1), pp. 2176.
(2024). YTHDF1 is pivotal for maintenance of cardiac homeostasis. Journal of Molecular and Cellular Cardiology, 193, pp. 25–35.
(2024). Nanopore Detection of METTL3-Dependent m6A-Modified mRNA Reveals a New Mechanism Regulating Cardiomyocyte Mitochondrial Metabolism. Circulation, 149(16), pp. 1319–1322.
(2024). In vitro inflammatory multi-cellular model of osteoarthritis. Osteoarthritis and Cartilage Open, 6(1), pp. 100432.
(2024). CITED4 gene therapy protects against maladaptive cardiac remodeling after ischemia/reperfusion injury in mice. Molecular Therapy: The Journal of the American Society of Gene Therapy, 32(10), pp. 3683–3694.
(2024). Cardiac cryptographers: cracking the code of the epitranscriptome. European Heart Journal, 45(23), pp. 2034–2036.
(2023). Loss of YTHDF2 Alters the Expression of m6A-Modified Myzap and Causes Adverse Cardiac Remodeling. JACC. Basic to translational science, 8(9), pp. 1180–1194.